Autoimmune ovarian disease: mechanism of disease induction and prevention
KSK Tung, YH Lou, KM Garza, C Teuscher - Current opinion in immunology, 1997 - Elsevier
KSK Tung, YH Lou, KM Garza, C Teuscher
Current opinion in immunology, 1997•ElsevierInvestigation of the versatile models for autoimmunity of the ovary and other selected organs
has contributed to our understanding of the following aspects of autoimmunity: the
mechanism of T cell molecular mimicry; T→ B epitope spreading, as a basis for autoantibody
diversification, and as a link between organ-specific and systemic autoimmunity; the
localization of genetic loci potentially influencing multiple autoimmune diseases; and the
elucidation of regulatory T cells as a component of physiological self tolerance.
has contributed to our understanding of the following aspects of autoimmunity: the
mechanism of T cell molecular mimicry; T→ B epitope spreading, as a basis for autoantibody
diversification, and as a link between organ-specific and systemic autoimmunity; the
localization of genetic loci potentially influencing multiple autoimmune diseases; and the
elucidation of regulatory T cells as a component of physiological self tolerance.
Investigation of the versatile models for autoimmunity of the ovary and other selected organs has contributed to our understanding of the following aspects of autoimmunity: the mechanism of T cell molecular mimicry; T→B epitope spreading, as a basis for autoantibody diversification, and as a link between organ-specific and systemic autoimmunity; the localization of genetic loci potentially influencing multiple autoimmune diseases; and the elucidation of regulatory T cells as a component of physiological self tolerance.
Elsevier